Atrioventricular Nodal Reentrant Tachycardia (AVNRT)
Epidemiology
Physiology
- Type of supraventricular tachycardia (SVT) characterized by AV-nodal reentry
- Both pathways reside within the AV node
 
 
Clinical
- Narrow-Complex Regular Tachycardia: usual manifestation
- Rate: usually >150
 
- Typical abrupt onset and termination
 
- Absence of readily identifiable p-waves
 
 
- Wide-Complex Regular Tachycardia: may be seen in cases with pre-existing bundle branch block or in cases with reta-related aberrant conduction
 
Treatment
- Stable: agents that inhibit AV nodal conduction
- Vagal Maneuvers (Carotid Massage, Valvsalva): when used alone, will terminate SVT’s in 25% of cases
 
- Adenosine
- Initial 6 mg via peripheral IV, followed by 20 ml flush
 
- Subsequent 6 mg via peripheral IV, followed by 20 ml flush
 
- Use initial dose 50% less if CVC, heart transplant, carbamazepine, dipyridamole
 
- Antagonized by theophylline, theobromine, or caffeine
 
- Side Effects: flushing, dyspnea/bronchospasm, chest discomfort
 
- Contraindications: asthma
 
- Pregnancy: safe
 
 
- Beta Blockers
 
- Calcium Channel Blockers
 
- Digoxin: less desirable
 
- Amiodarone
- Slower effect than adenosine
 
- Less desirable than AV nodal blockers in AVRT
 
 
 
- Unstable
- Synchronized cardioversion 50-100 J
 
 
References